Androgen Receptor Polyglutamine Repeat Length Affects Receptor Activity and C2c12 Cell Myogenic Potential

نویسندگان

  • Ryan L. Sheppard
  • Stephen M. Roth
  • Ryan Sheppard
چکیده

Title of Document: ANDROGEN RECEPTOR POLYGLUTAMINE REPEAT LENGTH AFFECTS RECEPTOR ACTIVITY AND C2C12 CELL MYOGENIC POTENTIAL

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CALL FOR PAPERS Functional Analysis of Sequence Variation Androgen receptor polyglutamine repeat length affects receptor activity and C2C12 cell development

Sheppard RL, Spangenburg EE, Chin ER, Roth SM. Androgen receptor polyglutamine repeat length affects receptor activity and C2C12 cell development. Physiol Genomics 43: 1135–1143, 2011. First published August 9, 2011; doi:10.1152/physiolgenomics.00049.2011.—Testosterone (T) has an anabolic effect on skeletal muscle and is believed to exert its local effects via the androgen receptor (AR). The AR...

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Androgen receptor polyglutamine repeat length affects receptor activity and C2C12 cell development.

Testosterone (T) has an anabolic effect on skeletal muscle and is believed to exert its local effects via the androgen receptor (AR). The AR harbors a polymorphic stretch of glutamine repeats demonstrated to inversely affect receptor transcriptional activity in prostate and kidney cells. The effects of AR glutamine repeat length on skeletal muscle are unknown. In this study we examined the effe...

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Background The androgen receptor (AR) gene contains a polymorphic trinucleotide repeat that encodes a polyglutamine tract in its N-terminal transactivation domain (NTAD). We aimed to find a correlation between the length of this polymorphic tract and azoospermia or oligozoospermia in infertile men living in Khuzestan, Iran. MaterialsAndMethods In this case-control study during two years till 20...

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Recruitment of the androgen receptor via serum response factor facilitates expression of a myogenic gene.

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Selective androgen receptor modulator, YK11, regulates myogenic differentiation of C2C12 myoblasts by follistatin expression.

The myogenic differentiation of C2C12 myoblast cells is induced by the novel androgen receptor (AR) partial agonist, (17α,20E)-17,20-[(1-methoxyethylidene)bis-(oxy)]-3-oxo-19-norpregna-4,20-diene-21-carboxylic acid methyl ester (YK11), as well as by dihydrotestosterone (DHT). YK11 is a selective androgen receptor modulator (SARM), which activates AR without the N/C interaction. In this study, w...

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تاریخ انتشار 2010